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At bicuculline tions b e t w e e n 0 . 0 fiM, A stimulation intensity B e v o k e d epileptiform ships revealed that these t w o c o m p o n e n t s had differ- activity a p p e a r e d . In contrast to w i d e s p r e a d p r o p a - ent c o n d u c t a n c e s and reversal potentials close to E i gation of epileptiform e v e n t s to all areas of the slice ( - 7 3 m V ) and E that o c c u r s with suprathreshold d o s e s of b i c u c u l l i n e , c o n d u c t a n c e s of C K ( - 8 9 m V ) , respectively (7).
20. Harris EW, Ganong AH, Cotman CW. Long-term potentiation in the hippocampus involves activation of yV-methyl-D-aspartate receptors. Brain Res 1984;323:132-7. 21. Heinemann U, Lux HD, Gutnick MJ. Extracellular free calcium and potassium during paroxsmal activity in the cerebral cortex of the cat. Exp Brain Res 1977;27:237-43. + 22. Hotson JR, Prince DA. Penicillin- and barium-induced epi+ leptiform bursting in hippocampal neurons: actions on C A and K potentials. Ann Neurol 1981;10:11-7. 23. Huguenard JR, Alger BE.
Ann Neurol 1977;1:463-9. 46. Schwartzkroin PA, Prince DA. Cellular and field potential properties of epileptogenic hippocampal slices. Brain Res 1978;147:117-30. 47. Schwartzkroin PA, Prince DA. Changes in excitatory and inhibitory synaptic potentials leading to epileptogenic activity. Brain Res 1980;183:61-76. 48. Segal M, Barker JL. Rat hippocampal neurons in culture: properties of GABA-activated C I ion conductance. J Neurophysiol 1984;51:500-15. 49. Sillito AM. Functional considerations of the operation of GABAergic inhibitory processes in the visual cortex.